Thursday, April 29, 2021

1601006181 General Medicine SHORT CASE


"This is an online e log book to discuss our patient’s de- identified health data shared after taking his/her/guardian’s signed informed consent. Here we discuss our individual patient’s problems through series of inputs from available global online community of experts with an aim to solve those patient’s clinical problems with collective current best evidence based inputs. This e-log book reflects my patient centered online portfolio and your valuable inputs on the comments is welcome.”

I have been given this case to solve in an attempt to understand the topic of “patient clinical data analysis” to develop my competency in reading and comprehending clinical data including history, clinical findings, investigations and come up with a diagnosis and treatment plan. 


 A 55 years old gentleman hailing from Miryalguda who is a labourer by occupation came to the hospital with the chief complaints of difficulty in breathing since 4 days.


HISTORY OF PRESENT ILLNESS:-

Patient was apparently asymptomatic 4 months ago. Then he developed fever which was:

  • Onset: insidious 
  • Nature: intermittent
  • Non progressive
  • Low grade
  • Not associated with chills and rigors
  • No diurnal variation 
  • Relieved on medication briefly 

As the fever was not subsiding he went to Miryalguda hospital where he was diagnosed to have Chronic Kidney Disease. He was prescribed medication for the same.


He developed Dyspnea on 21st April, 2021 Pitting edema around 2 am in night which was:

  • Onset- sudden
  • Associated with postural variation i.e. it increased on lying down (orthopnea)
  • Grade- 4 (According to NYHA classification)

He developed cough 3 days back which was::

  • Onset- sudden
  • Nature- dry
  • Non blood stained

There is no history of chest pain.

No history of palpitations.

No history of syncopal attacks.

No history of oliguria.

No history of hemoptysis.


He is a known case of hypertension since 5 years and is on medication.


He is a chronic alcoholic and a smoker since 18-20 years.

General examination: 

He was conscious, coherent, cooperative, moderately built and moderately nourished

He is well oriented to day, time, place, person

There is pallor

No icterus, cyanosis, clubbing, lymphadenopathy

There is Bilateral pitting Edema upto knees

    



Vitals:

  • He was afebrile.
  • Pulse rate: 92 bpm, regular in rhythm, normal in character and volume, no Radio- radial delay and no Radio-femoral delay.
  • Blood pressure: 180/100 mm Hg measured in right arm and in supine position.
  • Respiratory rate: 18 cpm
  • SpO2 at room air: 98%

Systemic examination:


1) Cardiovascular system:

JVP is elevated


Apex beat: well localised at the left 5th intercostal space half inch medial to mid clavicular line.



2) Respiratory system: Hyperpnea




Investigations done on this patient are:-


1) Renal Function Test:-



2) Hemogram

     


3) ECG


4) Chest X Ray


Treatment received by the patient:


1) Injection Lasix- loop diuretics

2) Tablet Nicardia- Calcium Channel Blocker

3) Injection erythropoietin

4) Tablet Nodosis - antacid

5) Tablet Shelcal

6) Tablet Orofex


He is undergoing dialysis


From the history, examination and investigations I am of the opinion that the patient has Chronic Kidney Disease due to chronic hypertension which is responsible for the-


1) Dyspnea

2) Pedal Edema

3) Anemia




 













Sunday, April 25, 2021

1601006181 GENERAL MEDICINE LONG CASE

 

This is an online e-log book to discuss our patient’s de- identified health data shared after taking his/her/guardian’s signed informed consent. Here we discuss our individual patient’s problems through series of inputs from available global online community of experts with an aim to solve those patient’s clinical problems with collective current best evidence based inputs. This e-log book reflects my patient centered online portfolio and your valuable inputs on the comments is welcome .

I have been given this case to solve in an attempt to understand the topic of “patient clinical data analysis” to develop my competency in reading and comprehending clinical data including history, clinical findings, investigations and come up with a diagnosis and treatment plan.


A 65 yr old woman hailing from Miryalguda who is a agricultural labourer by occupation came to the hospital on 23. 04 .21 with chief complaints of vomitings since 4 days


HISTORY OF PRESENTING ILLNESS: 

She is a known case of  type -2 diabetes since  10 yrs and hypertension since 1 yr and on medication for both

She was apparently asymptomatic 4 days back; then she developed vomitings which were :

  • Sudden in onset
  • 2 episodes /day
  • Pale in color
  • non projectile.
  • non bilious.
  • non blood stained
  • content:- food particles
  • immediately after eating food. 
  • Associated with constipation and belching since 3 days.

For which she went to miryalguda hospital &was referred to KIMS, Narketpally because of elevated serum creatinine (6.5 mg/dl) and blood urea (171 mg/dl)

She also had pedal edema 

  • Insidious in onset 
  • Since- 3 days
  • -pitting type


  •  limited to ankles. (grade-1) 


Negative  history

  • No history of fever ,loose stools,pain abdomen abdominal distension , headache, dyspepsia dysphagia
  • No h/o heart burn ,hematemesis meleana
  • No h/o oliguria , burning micturition  ,dysuria
  • No periorbital puffiness
  • No h/o Dyspnea, syncope ,palpitation , chest pain



PAST HISTORY

no history of  similar complaints in the past

no history of tb , bronchial asthma, epilepsy, thyroid abnormalities

PAST SURGICAL HISTORY ::

underwent hysterectomy 20years back

FAMILY HISTORY:

  • No similar complaints in other family members
  • No history of tuberculosis/ bronchial asthma / epilepsy/hypertension


PERSONAL HISTORY

  • diet: mixed
  • appetite: decreased
  • sleep: adequate
  • bladder movements : normal& bowel movements decreased 
  • no addictions


 MENSTRUAL HISTORY : 

attained menopause 20yrs back

children 3

DRUG HISTORY 

On medication for 

- Diabetes : Inj human actrapid insulin 200-x- 750 since 7 years


- Hypertension for 1 year 

  • Clindine trio
  • Cilndepine 10mg
  • Olmesartam 20 mg
  • Chlorthalidone 12.5 mg

- Pedal edema

  Torsemide 10 mg

No known drug allergies


TREATMENT HISTORY

1) IVF - NS

2)iv - lasix

3) iv-zofer 

4) iv - pantop

5) inj- human actrapid insulin HAI

6) pro-cremaffin


GENERAL EXAMINATION: 

  • informed consent  obtained 
  • conscious, coherent, cooperative
  • well oriented to day, date, time & place
  • moderately built & nourished


  • pallor- no signs of pallor
  • icterus - no signs of icterus
  • cyanosis - no signs of cyanosis
  • clubbing - absent
  • koilonychia - absent
  • lymadenopathy- absent
  • pedal edema - present

       


VITALS

  • Afebrile
  • Pulse Rate  : -70 bpm

- normal in character

-regular in rhythm

-no radio radial delay  radioofemoraldelay

-normal volume

  • BP: 160/80mm hg in left arm in supine position
  • RR:  20 cpm
  • SPO 2:  98% in room air


SYSTEMIC EXAMINATION

UPPER GIT EXAMINATION

Oral Cavity:

  • Lips     normal
  • teeth   normal
  • gums   normal
  • palate normal
  • oropharynx normal
  • cheek buccal  mucosa normal
  • tongue normal
  • breath   normal


Abdomen Examination::

Inspection

  • shape of abdomen : normal (scaphoid)


  • flanks : not full
  • no venous prominence
  • Umbilicus:  

      -central in position

      -normal (no discharge or hernia seen)

  • Skin over abdomen: 

      -no signs of scar ,sinuses ,ulcers

      -no pigmentation, striae

      -no localised swellings

      -no visible pulsations

  • movements of abdomen : all quadrants are moving with respiration
  • no visible peristalsis
  • hernial orifices: free


Palpation

Superficial:

  • no local rise of temperature
  • no tenderness
  • guarding  : absent
  • rigidity : absent


Deep Palpation : 

LIVER :

  • liver span: normal
  • no tenderness

SPLEEN : 

  • spleen not palpable

  • no rebound tenderness


Percussion 

  • Tympanic note heard; 
  • Dull note heard over the upper border of liver and spleen.
    • Liver span is normal   


    Auscultation 

    normal bowel sounds heard


    OTHER SYSTEMS

    CVS: 

    S1 S2 heard 

    JVP normal


    Respiratory system

    Normal vesicular breath sounds heard


    CNS

    Higher Mental functions : intact 

    Motor System : Reflexes preserved

    Babinskis sign : Flexor response

    Sensory system : Cranial nerves intact 


    From the history and examination, differential can be Acute GE, acute Gastritis, Bowel obstruction, endocrine or metabolic causes like gastroenteritis,gastric or bowel obstruction , Uremia etc.


    INVESTIGATIONS: 

    Hemogram

    CUE

    • Pus cells: 3-4
    • Epithelial cells: 2-4
    • RBCs: nil
    • Sugars: nil
    • Bile salts and bile pigments: nil 
    • Albumin: 2+
    • Spot urine Sodium: 153 mEq/L

    RFT

    • Serum urea: 199 mg/dl
    • Serum creatinine: 8.5 mg/dl
    • Serum uric acid: 7.8 mg/dl
    • Calcium: 10 mg/dl
    • Phosphorus: 4.7 mg/dl
    • Sodium: 135 mEq/L
    • Potassium: 4.2 mEq/L
    • Chloride: 106 mEq/L

    CXR

    USG ABDOMEN

                


    ECG




    From the investigations, I am of the opinion that it could be Acute Kidney Injury. The cause of her AKI could be-


    • Pre renal: Volume depletion from excessive vomitings 















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